Title : Interaction of the Anti-Proliferative GPER Inverse Agonist ERα17p with the Breast Cancer Cell Plasma Membrane: From Biophysics to Biology.

Pub. Date : 2020 Feb 15

PMID : 32075246






1 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 The peptide ERalpha17p, which corresponds to the 295-311 fragment of the hinge/AF2 domains of the human estrogen receptor alpha (ERalpha), exerts apoptosis in breast cancer cells through a mechanism involving the G protein-coupled estrogen-dependent receptor GPER. Estrogens estrogen receptor 1 Homo sapiens