Title : A Dietary Ketone Ester Normalizes Abnormal Behavior in a Mouse Model of Alzheimer's Disease.

Pub. Date : 2020 Feb 4

PMID : 32033248






4 Functional Relationships(s)
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Protein Name
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1 Because of a decreased sensitivity toward insulin, a key regulator of pyruvate dehydrogenase (PDH), Alzheimer"s patients have lower brain glucose utilization with reductions in Tricarboxylic Acid (TCA) cycle metabolites such as citrate, a precursor to n-acetyl-aspartate. Tricarboxylic Acids pyruvate dehydrogenase phosphatase catalytic subunit 1 Homo sapiens
2 Because of a decreased sensitivity toward insulin, a key regulator of pyruvate dehydrogenase (PDH), Alzheimer"s patients have lower brain glucose utilization with reductions in Tricarboxylic Acid (TCA) cycle metabolites such as citrate, a precursor to n-acetyl-aspartate. Tricarboxylic Acids pyruvate dehydrogenase phosphatase catalytic subunit 1 Homo sapiens
3 Because of a decreased sensitivity toward insulin, a key regulator of pyruvate dehydrogenase (PDH), Alzheimer"s patients have lower brain glucose utilization with reductions in Tricarboxylic Acid (TCA) cycle metabolites such as citrate, a precursor to n-acetyl-aspartate. Tricarboxylic Acids pyruvate dehydrogenase phosphatase catalytic subunit 1 Homo sapiens
4 Because of a decreased sensitivity toward insulin, a key regulator of pyruvate dehydrogenase (PDH), Alzheimer"s patients have lower brain glucose utilization with reductions in Tricarboxylic Acid (TCA) cycle metabolites such as citrate, a precursor to n-acetyl-aspartate. Tricarboxylic Acids pyruvate dehydrogenase phosphatase catalytic subunit 1 Homo sapiens