Title : Molecular cloning and the involvement of IRE1α-XBP1s signaling pathway in palmitic acid induced - Inflammation in primary hepatocytes from large yellow croaker (Larimichthys crocea).

Pub. Date : 2020 Mar

PMID : 31904542






4 Functional Relationships(s)
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1 The ER stress-inducing agent tunicamycin (Tm) and PA treatment significantly activated the IRE1alpha-XBP1s signaling pathway and increased the pro-inflammatory genes expression including tumor necrosis factor alpha (tnfalpha), interleukin 6 (il-6) and interleukin 1beta (il-1beta) (P < 0.05). Tunicamycin tumor necrosis factor b (TNF superfamily, member 2) Larimichthys crocea
2 The ER stress-inducing agent tunicamycin (Tm) and PA treatment significantly activated the IRE1alpha-XBP1s signaling pathway and increased the pro-inflammatory genes expression including tumor necrosis factor alpha (tnfalpha), interleukin 6 (il-6) and interleukin 1beta (il-1beta) (P < 0.05). Tunicamycin tumor necrosis factor b (TNF superfamily, member 2) Larimichthys crocea
3 The ER stress-inducing agent tunicamycin (Tm) and PA treatment significantly activated the IRE1alpha-XBP1s signaling pathway and increased the pro-inflammatory genes expression including tumor necrosis factor alpha (tnfalpha), interleukin 6 (il-6) and interleukin 1beta (il-1beta) (P < 0.05). Tunicamycin tumor necrosis factor b (TNF superfamily, member 2) Larimichthys crocea
4 The ER stress-inducing agent tunicamycin (Tm) and PA treatment significantly activated the IRE1alpha-XBP1s signaling pathway and increased the pro-inflammatory genes expression including tumor necrosis factor alpha (tnfalpha), interleukin 6 (il-6) and interleukin 1beta (il-1beta) (P < 0.05). Tunicamycin tumor necrosis factor b (TNF superfamily, member 2) Larimichthys crocea