Title : Exenatide induces frataxin expression and improves mitochondrial function in Friedreich ataxia.

Pub. Date : 2020 Jan 30

PMID : 31877117






2 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Friedreich ataxia is caused by intronic GAA trinucleotide repeat expansions in the frataxin-encoding FXN gene that reduce frataxin expression, impair iron-sulfur cluster biogenesis, cause oxidative stress, and result in mitochondrial dysfunction and apoptosis. Iron frataxin Homo sapiens
2 Friedreich ataxia is caused by intronic GAA trinucleotide repeat expansions in the frataxin-encoding FXN gene that reduce frataxin expression, impair iron-sulfur cluster biogenesis, cause oxidative stress, and result in mitochondrial dysfunction and apoptosis. Iron frataxin Homo sapiens