Title : PI3K/AKT/mTOR signaling as a molecular target in head and neck cancer.

Pub. Date : 2020 Feb

PMID : 31785230






1 Functional Relationships(s)
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1 PI3K/AKT/mTOR signaling is active in over 90% of HNSCC, as a result of EGFR activation (47%), phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha (PIK3CA) mutations (8.6%), PIK3CA amplifications (14.2%), phosphatidylinositol 3-kinase (PI3K) overexpression (27.2%) and phosphatase and tensin homolog (PTEN) mutation (10-15%). phosphatidylinositol 3,5-diphosphate AKT serine/threonine kinase 1 Homo sapiens