Title : PAR2 controls cholesterol homeostasis and lipid metabolism in nonalcoholic fatty liver disease.

Pub. Date : 2019 Nov

PMID : 31668396






3 Functional Relationships(s)
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1 Conversely, PAR2 deficiency in mice resulted in reduced expression of key hepatic genes involved in cholesterol synthesis, a 50% drop in plasma and total liver cholesterol, and induced a reverse cholesterol transport system that culminated in 25% higher fecal bile acid output. Cholesterol pulmonary adenoma resistance 2 Mus musculus
2 Conversely, PAR2 deficiency in mice resulted in reduced expression of key hepatic genes involved in cholesterol synthesis, a 50% drop in plasma and total liver cholesterol, and induced a reverse cholesterol transport system that culminated in 25% higher fecal bile acid output. Cholesterol pulmonary adenoma resistance 2 Mus musculus
3 Conversely, PAR2 deficiency in mice resulted in reduced expression of key hepatic genes involved in cholesterol synthesis, a 50% drop in plasma and total liver cholesterol, and induced a reverse cholesterol transport system that culminated in 25% higher fecal bile acid output. Cholesterol pulmonary adenoma resistance 2 Mus musculus