Title : Dysregulation of Gap Junction Function and Cytokine Production in Response to Non-Genotoxic Polycyclic Aromatic Hydrocarbons in an In Vitro Lung Cell Model.

Pub. Date : 2019 Apr 23

PMID : 31018556






1 Functional Relationships(s)
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1 Parthenolide, a pan-inflammation inhibitor, reversed the PAH-induced inhibition of GJIC, the decreased CX43 expression, and the induction of KC and TNF. Polycyclic Aromatic Hydrocarbons gap junction protein, alpha 1 Mus musculus