Title : Epigallocatechin-3-gallate inhibits doxorubicin-induced inflammation on human ovarian tissue.

Pub. Date : 2019 May 31

PMID : 30996116






2 Functional Relationships(s)
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1 DOX influenced inflammatory responses by inducing a significant increase in the expression of pro-inflammatory cytokines, such as tumor necrosis factor-alpha (TNF-alpha) and cyclooxigenase-2 (COX-2), of inflammatory interleukins (IL), such as interleukin-6 (IL-6) and interleukin-8 (IL-8), and the inflammatory proteins mediators metalloproteinase-2 and metalloproteinase-9 (MMP2 and MMP9). Doxorubicin tumor necrosis factor Homo sapiens
2 DOX influenced inflammatory responses by inducing a significant increase in the expression of pro-inflammatory cytokines, such as tumor necrosis factor-alpha (TNF-alpha) and cyclooxigenase-2 (COX-2), of inflammatory interleukins (IL), such as interleukin-6 (IL-6) and interleukin-8 (IL-8), and the inflammatory proteins mediators metalloproteinase-2 and metalloproteinase-9 (MMP2 and MMP9). Doxorubicin tumor necrosis factor Homo sapiens