Title : Metabolic dependence of cyclosporine A on cell proliferation of human non‑small cell lung cancer A549 cells and its implication in post‑transplant malignancy.

Pub. Date : 2019 May

PMID : 30896878






2 Functional Relationships(s)
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1 Mechanistically, CsA treatment increased reactive oxygen species (ROS) generation, and the intracellular ROS scavenger N-acetyl-cysteine (NAC) attenuated CsA-induced cell proliferation as well as the activation of Akt/Cyclin D1 signaling. Acetylcysteine AKT serine/threonine kinase 1 Homo sapiens
2 Mechanistically, CsA treatment increased reactive oxygen species (ROS) generation, and the intracellular ROS scavenger N-acetyl-cysteine (NAC) attenuated CsA-induced cell proliferation as well as the activation of Akt/Cyclin D1 signaling. Acetylcysteine AKT serine/threonine kinase 1 Homo sapiens