Title : Imbalanced sphingolipid signaling is maintained as a core proponent of a cancerous phenotype in spite of metabolic pressure and epigenetic drift.

Pub. Date : 2019 Jan 11

PMID : 30728898






1 Functional Relationships(s)
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1 Relative to hTert-HPNE, PSN-1 subclones uniformly maintained modified sphingolipid signaling and specifically retained elevated sphingosine-1-phosphate (S1P) relative to C16 ceramide (C16 Cer) ratios. Sphingolipids 5'-nucleotidase, cytosolic IIIA Homo sapiens