Title : AKT methylation by SETDB1 promotes AKT kinase activity and oncogenic functions.

Pub. Date : 2019 Feb

PMID : 30692625






3 Functional Relationships(s)
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1 Mechanistically, the interaction of phosphatidylinositol (3,4,5)-trisphosphate with AKT facilitates its interaction with SETDB1 for subsequent AKT methylation, which in turn sustains AKT phosphorylation. phosphatidylinositol 3,4,5-triphosphate thymoma viral proto-oncogene 1 Mus musculus
2 Mechanistically, the interaction of phosphatidylinositol (3,4,5)-trisphosphate with AKT facilitates its interaction with SETDB1 for subsequent AKT methylation, which in turn sustains AKT phosphorylation. phosphatidylinositol 3,4,5-triphosphate thymoma viral proto-oncogene 1 Mus musculus
3 Mechanistically, the interaction of phosphatidylinositol (3,4,5)-trisphosphate with AKT facilitates its interaction with SETDB1 for subsequent AKT methylation, which in turn sustains AKT phosphorylation. phosphatidylinositol 3,4,5-triphosphate thymoma viral proto-oncogene 1 Mus musculus