Title : Common ELF1 deletion in prostate cancer bolsters oncogenic ETS function, inhibits senescence and promotes docetaxel resistance.

Pub. Date : 2018 May

PMID : 30603056






2 Functional Relationships(s)
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1 Common ELF1 deletion in prostate cancer bolsters oncogenic ETS function, inhibits senescence and promotes docetaxel resistance. Docetaxel E74 like ETS transcription factor 1 Homo sapiens
2 Furthermore, knockdown of ELF1 increased docetaxel resistance, indicating that the genomic deletions found in metastatic prostate tumors may promote therapeutic resistance through loss of both RB1 and ELF1. Docetaxel E74 like ETS transcription factor 1 Homo sapiens