Title : Excitotoxic superoxide production and neuronal death require both ionotropic and non-ionotropic NMDA receptor signaling.

Pub. Date : 2018 Nov 30

PMID : 30504838






3 Functional Relationships(s)
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1 Neurons expressing either GluN2B subunits or chimeric GluN2A/GluN2B C-terminus subunits exhibited NMDA-induced superoxide production, whereas neurons expressing chimeric GluN2B/GluN2A C-terminus subunits did not. Superoxides glutamate receptor, ionotropic, NMDA2B (epsilon 2) Mus musculus
2 Neurons expressing either GluN2B subunits or chimeric GluN2A/GluN2B C-terminus subunits exhibited NMDA-induced superoxide production, whereas neurons expressing chimeric GluN2B/GluN2A C-terminus subunits did not. Superoxides glutamate receptor, ionotropic, NMDA2B (epsilon 2) Mus musculus
3 Neurons expressing either GluN2B subunits or chimeric GluN2A/GluN2B C-terminus subunits exhibited NMDA-induced superoxide production, whereas neurons expressing chimeric GluN2B/GluN2A C-terminus subunits did not. Superoxides glutamate receptor, ionotropic, NMDA2B (epsilon 2) Mus musculus