Title : Th1 cytokines TNF-α and IFN-γ promote corticosteroid resistance in developing human airway smooth muscle.

Pub. Date : 2019 Jan 1

PMID : 30335498






4 Functional Relationships(s)
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1 In contrast to TNF-alpha or IFN-gamma alone, the combination of TNF-alpha/IFN-gamma blunted the ability of fluticasone propionate (FP) to reduce expression of the chemokines CCL5 and CXCL10 despite expression of key anti-inflammatory glucocorticoid receptor target genes being largely unaffected by TNF-alpha/IFN-gamma. Fluticasone tumor necrosis factor Homo sapiens
2 In contrast to TNF-alpha or IFN-gamma alone, the combination of TNF-alpha/IFN-gamma blunted the ability of fluticasone propionate (FP) to reduce expression of the chemokines CCL5 and CXCL10 despite expression of key anti-inflammatory glucocorticoid receptor target genes being largely unaffected by TNF-alpha/IFN-gamma. Fluticasone tumor necrosis factor Homo sapiens
3 In contrast to TNF-alpha or IFN-gamma alone, the combination of TNF-alpha/IFN-gamma blunted the ability of fluticasone propionate (FP) to reduce expression of the chemokines CCL5 and CXCL10 despite expression of key anti-inflammatory glucocorticoid receptor target genes being largely unaffected by TNF-alpha/IFN-gamma. Fluticasone tumor necrosis factor Homo sapiens
4 In contrast to TNF-alpha or IFN-gamma alone, the combination of TNF-alpha/IFN-gamma blunted the ability of fluticasone propionate (FP) to reduce expression of the chemokines CCL5 and CXCL10 despite expression of key anti-inflammatory glucocorticoid receptor target genes being largely unaffected by TNF-alpha/IFN-gamma. Fluticasone tumor necrosis factor Homo sapiens