Pub. Date : 2019 Feb
PMID : 30187510
8 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | Effects of bisphenol A on gap junctions in HaCaT cells as mediated by the estrogen receptor pathway. | bisphenol A | estrogen receptor 1 | Homo sapiens |
| 2 | Estrogen receptor (ER) antagonist ICI 182 780 at 5 nm partially blocked the above effects of BPA indicating involvement of the ER pathway with BPA exposure. | bisphenol A | estrogen receptor 1 | Homo sapiens |
| 3 | Estrogen receptor (ER) antagonist ICI 182 780 at 5 nm partially blocked the above effects of BPA indicating involvement of the ER pathway with BPA exposure. | bisphenol A | estrogen receptor 1 | Homo sapiens |
| 4 | Estrogen receptor (ER) antagonist ICI 182 780 at 5 nm partially blocked the above effects of BPA indicating involvement of the ER pathway with BPA exposure. | bisphenol A | estrogen receptor 1 | Homo sapiens |
| 5 | Estrogen receptor (ER) antagonist ICI 182 780 at 5 nm partially blocked the above effects of BPA indicating involvement of the ER pathway with BPA exposure. | bisphenol A | estrogen receptor 1 | Homo sapiens |
| 6 | Estrogen receptor (ER) antagonist ICI 182 780 at 5 nm partially blocked the above effects of BPA indicating involvement of the ER pathway with BPA exposure. | bisphenol A | estrogen receptor 1 | Homo sapiens |
| 7 | Estrogen receptor (ER) antagonist ICI 182 780 at 5 nm partially blocked the above effects of BPA indicating involvement of the ER pathway with BPA exposure. | bisphenol A | estrogen receptor 1 | Homo sapiens |
| 8 | In conclusion, our results indicate that BPA can promote human skin cell proliferation, inhibit skin cell GJIC function but not formation and downregulate Cx26 mRNA levels partially through the ER pathway. | bisphenol A | estrogen receptor 1 | Homo sapiens |