Title : Mitochondrial ROS activate interleukin-1β expression in allergic rhinitis.

Pub. Date : 2018 Sep

PMID : 30127914






1 Functional Relationships(s)
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1 The results of the present study suggested that patients with AR have raised mitochondrial ROS levels, which may upregulate the expression of IL-1beta, affecting IL-17-production and serving a role in the pathogenesis of AR. Reactive Oxygen Species interleukin 17A Homo sapiens