Title : PIM2 survival kinase is upregulated in a p53-dependent manner in cells treated with camptothecin or co-treated with actinomycin D and nutlin-3a.

Pub. Date : 2018 Oct 1

PMID : 30096294






3 Functional Relationships(s)
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1 The accumulation of PIM2 following treatment with A + N was suppressed in p53-knockdown cells. Ethanol tumor protein p53 Homo sapiens
2 Moreover, we found that in A549 cells, the treatment with A + N stimulated in p53-dependent fashion the expression of other high cooperativity p53 target genes, DRAXIN and H19. Ethanol tumor protein p53 Homo sapiens
3 Moreover, we found that in A549 cells, the treatment with A + N stimulated in p53-dependent fashion the expression of other high cooperativity p53 target genes, DRAXIN and H19. Ethanol tumor protein p53 Homo sapiens