Title : Hepatocyte-Specific Deletion of TIPARP, a Negative Regulator of the Aryl Hydrocarbon Receptor, Is Sufficient to Increase Sensitivity to Dioxin-Induced Wasting Syndrome.

Pub. Date : 2018 Oct 1

PMID : 29873790






3 Functional Relationships(s)
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1 We previously reported that loss of TCDD-inducible poly(adenosine diphosphate [ADP]-ribose) polymerase (TIPARP/PARP7/ARTD14), an AHR target gene and mono-ADP-ribosyltransferase, increases the sensitivity of mice to dioxin-induced toxicities. Polychlorinated Dibenzodioxins TCDD-inducible poly(ADP-ribose) polymerase Mus musculus
2 We previously reported that loss of TCDD-inducible poly(adenosine diphosphate [ADP]-ribose) polymerase (TIPARP/PARP7/ARTD14), an AHR target gene and mono-ADP-ribosyltransferase, increases the sensitivity of mice to dioxin-induced toxicities. Polychlorinated Dibenzodioxins TCDD-inducible poly(ADP-ribose) polymerase Mus musculus
3 We previously reported that loss of TCDD-inducible poly(adenosine diphosphate [ADP]-ribose) polymerase (TIPARP/PARP7/ARTD14), an AHR target gene and mono-ADP-ribosyltransferase, increases the sensitivity of mice to dioxin-induced toxicities. Polychlorinated Dibenzodioxins TCDD-inducible poly(ADP-ribose) polymerase Mus musculus