Title : Impaired lipid metabolism markers to assess the risk of neuroinflammation in autism spectrum disorder.

Pub. Date : 2018 Aug

PMID : 29569150






3 Functional Relationships(s)
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1 Based on the discussed data, the positive correlation between PGE2, COX-2, and mPGES-1 confirm the role of PGE2 pathway and neuroinflammation in the etiology of ASD, and the possibility of using PGE2, COX-2 and mPGES-1 as biomarkers of autism severity. Dinoprostone prostaglandin E synthase Mus musculus
2 Based on the discussed data, the positive correlation between PGE2, COX-2, and mPGES-1 confirm the role of PGE2 pathway and neuroinflammation in the etiology of ASD, and the possibility of using PGE2, COX-2 and mPGES-1 as biomarkers of autism severity. Dinoprostone prostaglandin E synthase Mus musculus
3 Based on the discussed data, the positive correlation between PGE2, COX-2, and mPGES-1 confirm the role of PGE2 pathway and neuroinflammation in the etiology of ASD, and the possibility of using PGE2, COX-2 and mPGES-1 as biomarkers of autism severity. Dinoprostone prostaglandin E synthase Mus musculus