Title : Involvement of the cystathionine-γ-lyase/Cav3.2 pathway in substance P-induced bladder pain in the mouse, a model for nonulcerative bladder pain syndrome.

Pub. Date : 2018 May 1

PMID : 29407215






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Hydrogen sulfide (H2S) formed by cystathionine-gamma-lyase (CSE) enhances the activity of Cav3.2 T-type Ca2+ channels, contributing to the bladder pain accompanying hemorrhagic cystitis caused by systemic administration of cyclophosphamide (CPA) in mice. Cyclophosphamide calcium channel, voltage-dependent, T type, alpha 1H subunit Mus musculus
2 Hydrogen sulfide (H2S) formed by cystathionine-gamma-lyase (CSE) enhances the activity of Cav3.2 T-type Ca2+ channels, contributing to the bladder pain accompanying hemorrhagic cystitis caused by systemic administration of cyclophosphamide (CPA) in mice. Cyclophosphamide calcium channel, voltage-dependent, T type, alpha 1H subunit Mus musculus