Title : ATM Is Required for the Prolactin-Induced HSP90-Mediated Increase in Cellular Viability and Clonogenic Growth After DNA Damage.

Pub. Date : 2018 Feb 1

PMID : 29186352






2 Functional Relationships(s)
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1 Inhibitors of JAK2 (G6) and ATM (KU55933) abolished the PRL-mediated increase in cell viability of DNA-damaged cells, supporting the involvement of each, as well as the crosstalk of ATM with the PRL pathway in the context of DNA damage. 2-morpholin-4-yl-6-thianthren-1-yl-pyran-4-one ATM serine/threonine kinase Homo sapiens
2 Inhibitors of JAK2 (G6) and ATM (KU55933) abolished the PRL-mediated increase in cell viability of DNA-damaged cells, supporting the involvement of each, as well as the crosstalk of ATM with the PRL pathway in the context of DNA damage. 2-morpholin-4-yl-6-thianthren-1-yl-pyran-4-one ATM serine/threonine kinase Homo sapiens