Title : Hydrogen sulfide ameliorates subarachnoid hemorrhage-induced neuronal apoptosis via the ROS-MST1 pathway.

Pub. Date : 2017 Sep 26

PMID : 29088725






4 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Western blot analysis demonstrated that exogenous NaHS decreased cleaved MST1 (cl-MST1) while increasing full-length MST1 expression. sodium bisulfide macrophage stimulating 1 Homo sapiens
2 Western blot analysis demonstrated that exogenous NaHS decreased cleaved MST1 (cl-MST1) while increasing full-length MST1 expression. sodium bisulfide macrophage stimulating 1 Homo sapiens
3 Western blot analysis demonstrated that exogenous NaHS decreased cleaved MST1 (cl-MST1) while increasing full-length MST1 expression. sodium bisulfide macrophage stimulating 1 Homo sapiens
4 CONCLUSIONS: Exogenous NaHS, as a donor of H2S, could ameliorate early brain injury after SAH by inhibiting neuronal apoptosis by reducing the activity of the MST1 protein. sodium bisulfide macrophage stimulating 1 Homo sapiens