Title : Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.

Pub. Date : 2018 Jan

PMID : 29042083






1 Functional Relationships(s)
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1 Treatment of nephrotic mice with the serine protease inhibitor aprotinin by means of subcutaneous sustained-release pellets normalized urinary serine protease activity and prevented sodium retention, as did treatment with the ENaC inhibitor amiloride. Amiloride sodium channel, nonvoltage-gated 1 alpha Mus musculus