Title : Persistent antidepressant effect of low-dose ketamine and activation in the supplementary motor area and anterior cingulate cortex in treatment-resistant depression: A randomized control study.

Pub. Date : 2018 Jan 1

PMID : 28922734






7 Functional Relationships(s)
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1 The increase in the SUV in the dACC was negatively correlated with depressive symptoms at 1day after ketamine infusion DISCUSSION: The persistent antidepressant effect of a 0.5mg/kg ketamine infusion may be mediated by increased activation in the SMA and dACC. Ketamine Acetyl-CoA carboxylase Drosophila melanogaster
2 The increase in the SUV in the dACC was negatively correlated with depressive symptoms at 1day after ketamine infusion DISCUSSION: The persistent antidepressant effect of a 0.5mg/kg ketamine infusion may be mediated by increased activation in the SMA and dACC. Ketamine Acetyl-CoA carboxylase Drosophila melanogaster
3 The increase in the SUV in the dACC was negatively correlated with depressive symptoms at 1day after ketamine infusion DISCUSSION: The persistent antidepressant effect of a 0.5mg/kg ketamine infusion may be mediated by increased activation in the SMA and dACC. Ketamine Acetyl-CoA carboxylase Drosophila melanogaster
4 The increase in the SUV in the dACC was negatively correlated with depressive symptoms at 1day after ketamine infusion DISCUSSION: The persistent antidepressant effect of a 0.5mg/kg ketamine infusion may be mediated by increased activation in the SMA and dACC. Ketamine Acetyl-CoA carboxylase Drosophila melanogaster
5 The higher increase in dACC activation was related to the reduction in depressive symptoms after ketamine infusion. Ketamine Acetyl-CoA carboxylase Drosophila melanogaster
6 A 0.5mg/kg ketamine infusion facilitated the glutamatergic neurotransmission in the SMA and dACC, which may be responsible for the persistent antidepressant effect of ketamine much beyond its half-life. Ketamine Acetyl-CoA carboxylase Drosophila melanogaster
7 A 0.5mg/kg ketamine infusion facilitated the glutamatergic neurotransmission in the SMA and dACC, which may be responsible for the persistent antidepressant effect of ketamine much beyond its half-life. Ketamine Acetyl-CoA carboxylase Drosophila melanogaster