Title : LAV-BPIFB4 isoform modulates eNOS signalling through Ca2+/PKC-alpha-dependent mechanism.

Pub. Date : 2017 Jun 1

PMID : 28419216






1 Functional Relationships(s)
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1 In addition, although LAV-BPIFB4 lost the ability to activate PKCalpha and eNOS in ex vivo vessels studied in an external Ca2+-free medium and in vessels from eNOS-/- mice, it still potentiated endothelial activity, recruiting an alternative mechanism dependent upon endothelium-derived hyperpolarizing factor (EDHF). edhf BPI fold containing family B, member 4 Mus musculus