Title : Argon Exposure Induces Postconditioning in Myocardial Ischemia-Reperfusion.

Pub. Date : 2017 Nov

PMID : 28381122






1 Functional Relationships(s)
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1 This effect of argon was abolished in the presence of wortmannin and PD98059 which inhibit prosurvival phosphatidylinositol-3-kinase (PI3K)/protein kinase B (Akt) and MEK/extracellular receptor kinase 1/2 (ERK 1/2), respectively, or in the presence of the mitochondrial permeability transition pore opener atractyloside, suggesting the involvement of the reperfusion injury salvage kinase pathway. Wortmannin AKT serine/threonine kinase 1 Homo sapiens