Title : Early Development of Parvalbumin-, Somatostatin-, and Cholecystokinin-Expressing Neurons in Rat Brain following Prenatal Immune Activation and Maternal Iron Deficiency.

Pub. Date : 2016

PMID : 28214898






3 Functional Relationships(s)
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1 Substantial evidence suggests that abnormalities in inhibitory gamma-aminobutyric acid (GABA) interneuron function, especially in the parvalbumin subtype of GABA interneuron, both developmentally and in adulthood, may contribute mechanistically to cognitive deficits and psychotic symptoms in schizophrenia. gamma-Aminobutyric Acid parvalbumin Rattus norvegicus
2 Substantial evidence suggests that abnormalities in inhibitory gamma-aminobutyric acid (GABA) interneuron function, especially in the parvalbumin subtype of GABA interneuron, both developmentally and in adulthood, may contribute mechanistically to cognitive deficits and psychotic symptoms in schizophrenia. gamma-Aminobutyric Acid parvalbumin Rattus norvegicus
3 Substantial evidence suggests that abnormalities in inhibitory gamma-aminobutyric acid (GABA) interneuron function, especially in the parvalbumin subtype of GABA interneuron, both developmentally and in adulthood, may contribute mechanistically to cognitive deficits and psychotic symptoms in schizophrenia. gamma-Aminobutyric Acid parvalbumin Rattus norvegicus