Title : TP53 and Decitabine in Acute Myeloid Leukemia and Myelodysplastic Syndromes.

Pub. Date : 2016 Nov 24

PMID : 27959731






1 Functional Relationships(s)
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1 CONCLUSIONS: Patients with AML and MDS who had cytogenetic abnormalities associated with unfavorable risk, TP53 mutations, or both had favorable clinical responses and robust (but incomplete) mutation clearance after receiving serial 10-day courses of decitabine. Decitabine tumor protein p53 Homo sapiens