Title : LRRC8A channels support TNF╬▒-induced superoxide production by Nox1 which is required for receptor endocytosis.

Pub. Date : 2016 Dec

PMID : 27838438






5 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 LRRC8A channels support TNFalpha-induced superoxide production by Nox1 which is required for receptor endocytosis. Superoxides tumor necrosis factor Homo sapiens
2 In vascular smooth muscle cells, tumor necrosis factor-alpha (TNFalpha) activates VRAC via type 1 TNFalpha receptors (TNFR1), and this requires superoxide (O2 -) production by NADPH oxidase 1 (Nox1). Superoxides tumor necrosis factor Homo sapiens
3 In vascular smooth muscle cells, tumor necrosis factor-alpha (TNFalpha) activates VRAC via type 1 TNFalpha receptors (TNFR1), and this requires superoxide (O2 -) production by NADPH oxidase 1 (Nox1). Superoxides tumor necrosis factor Homo sapiens
4 In vascular smooth muscle cells, tumor necrosis factor-alpha (TNFalpha) activates VRAC via type 1 TNFalpha receptors (TNFR1), and this requires superoxide (O2 -) production by NADPH oxidase 1 (Nox1). Superoxides tumor necrosis factor Homo sapiens
5 In vascular smooth muscle cells, tumor necrosis factor-alpha (TNFalpha) activates VRAC via type 1 TNFalpha receptors (TNFR1), and this requires superoxide (O2 -) production by NADPH oxidase 1 (Nox1). Superoxides tumor necrosis factor Homo sapiens