Title : Monomeric adiponectin increases cell viability in porcine aortic endothelial cells cultured in normal and high glucose conditions: Data on kinases activation.

Pub. Date : 2016 Sep

PMID : 27583345






5 Functional Relationships(s)
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1 Monomeric adiponectin increases cell viability in porcine aortic endothelial cells cultured in normal and high glucose conditions: Data on kinases activation. Glucose adiponectin, C1Q and collagen domain containing Homo sapiens
2 We found that monomeric adiponectin was able to increase cell viability in porcine aortic endothelial cells (PAE) cultured both in normal and high glucose condition. Glucose adiponectin, C1Q and collagen domain containing Homo sapiens
3 Moreover, in normal glucose condition monomeric adiponectin increased p38MAPK, Akt, ERK1/2 and eNOS phosphorylation in a dose- and time-dependent way. Glucose adiponectin, C1Q and collagen domain containing Homo sapiens
4 Also in high glucose condition monomeric adiponectin increased eNOS and above kinases phosphorylation with similar patterns but at lower extent. Glucose adiponectin, C1Q and collagen domain containing Homo sapiens
5 For interpretation of the data presented in this article, please see the research article "Monomeric adiponectin modulates nitric oxide release and calcium movements in porcine aortic endothelial cells in normal/high glucose conditions" (Grossini et al., in press) [1]. Glucose adiponectin, C1Q and collagen domain containing Homo sapiens