Title : Enhancing doxorubicin efficacy through inhibition of aspartate transaminase in triple-negative breast cancer cells.

Pub. Date : 2016 May 13

PMID : 27086848






1 Functional Relationships(s)
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1 Following doxorubicin administration, TNBC cells acquire metabolic alteration, causing increased glutamine flux for the synthesis of aspartate which can be converted into OAA by GOT1. Aspartic Acid glutamic-oxaloacetic transaminase 1 Homo sapiens