Title : Potential identification of a claudicant group vulnerable to acute kidney injury.

Pub. Date : 2016 Oct

PMID : 26980004






4 Functional Relationships(s)
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1 Of the 20 participants in this study, the urinary NAG/creatinine (Cr) rose from a pre-exercise level of 8.9, 6.7 to 14.3 (mumol/L/mmol Cr; median, IQR) to 12.9, 9.1 to 17.7 post exercise (p = 0.0003, Mann Whitney U test). Chromium N-acetyl-alpha-glucosaminidase Homo sapiens
2 Of the 20 participants in this study, the urinary NAG/creatinine (Cr) rose from a pre-exercise level of 8.9, 6.7 to 14.3 (mumol/L/mmol Cr; median, IQR) to 12.9, 9.1 to 17.7 post exercise (p = 0.0003, Mann Whitney U test). Chromium N-acetyl-alpha-glucosaminidase Homo sapiens
3 Participants with a rise in the NAG/Cr ratio post exercise had a greater ability to increase endothelial reactivity (%; median, IQR; 2.56, 0.1 to 3.7) cf. Chromium N-acetyl-alpha-glucosaminidase Homo sapiens
4 The mechanism by which the phenotypically distinct sub-group of patients with intermittent claudication who experience a NAG/Cr rise involves complex interactions between systemic oxidative stress and endothelial function. Chromium N-acetyl-alpha-glucosaminidase Homo sapiens