Title : Melatonin enhances arsenic trioxide-induced cell death via sustained upregulation of Redd1 expression in breast cancer cells.

Pub. Date : 2016 Feb 15

PMID : 26607805






1 Functional Relationships(s)
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1 Melatonin enhanced the ATO-induced apoptotic cell death via changes in the protein levels of Survivin, Bcl-2, and Bax, thus affecting cytochrome c release from the mitochondria to the cytosol. Arsenic Trioxide BCL2 associated X, apoptosis regulator Homo sapiens