Title : Inhibition of glutamine utilization sensitizes lung cancer cells to apigenin-induced apoptosis resulting from metabolic and oxidative stress.

Pub. Date : 2016 Jan

PMID : 26573871






1 Functional Relationships(s)
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1 The underlying mechanisms were defined that the anticancer effects of apigenin were reversed by ectopic GLUT1 overexpression and galactose supplementation, through activation of pentose phosphate pathway-mediated NADPH generation. Pentosephosphates solute carrier family 2 (facilitated glucose transporter), member 1 Mus musculus