Title : Loss of NOX-Derived Superoxide Exacerbates Diabetogenic CD4 T-Cell Effector Responses in Type 1 Diabetes.

Pub. Date : 2015 Dec

PMID : 26269022

1 Functional Relationships(s)
Compound Name
Protein Name
1 Our laboratory recently demonstrated that superoxide-deficient nonobese diabetic (NOD.Ncf1(m1J)) mice exhibited a delay in type 1 diabetes (T1D) partially due to blunted IFN-gamma synthesis by CD4 T cells. Superoxides interferon gamma Mus musculus