Title : Akt kinase C-terminal modifications control activation loop dephosphorylation and enhance insulin response.

Pub. Date : 2015 Oct 1

PMID : 26201515






1 Functional Relationships(s)
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1 Furthermore, the phosphatase-resistant Akt was refractory to ceramide-dependent dephosphorylation and amplified insulin-dependent Thr(308) phosphorylation in a regulated fashion. Ceramides AKT serine/threonine kinase 1 Homo sapiens