Title : Cardiac-specific ablation of the STAT3 gene in the subacute phase of myocardial infarction exacerbated cardiac remodeling.

Pub. Date : 2015 Aug 1

PMID : 26055795






6 Functional Relationships(s)
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1 In the present study, we assessed the biological significance of STAT3 activity in subacute MI using tamoxifen (TM)-inducible cardiac-specific STAT3 knockout (STAT3 iCKO) mice. Tamoxifen signal transducer and activator of transcription 3 Mus musculus
2 In the present study, we assessed the biological significance of STAT3 activity in subacute MI using tamoxifen (TM)-inducible cardiac-specific STAT3 knockout (STAT3 iCKO) mice. Tamoxifen signal transducer and activator of transcription 3 Mus musculus
3 In the present study, we assessed the biological significance of STAT3 activity in subacute MI using tamoxifen (TM)-inducible cardiac-specific STAT3 knockout (STAT3 iCKO) mice. Tamoxifen signal transducer and activator of transcription 3 Mus musculus
4 In the present study, we assessed the biological significance of STAT3 activity in subacute MI using tamoxifen (TM)-inducible cardiac-specific STAT3 knockout (STAT3 iCKO) mice. Tamoxifen signal transducer and activator of transcription 3 Mus musculus
5 In the present study, we assessed the biological significance of STAT3 activity in subacute MI using tamoxifen (TM)-inducible cardiac-specific STAT3 knockout (STAT3 iCKO) mice. Tamoxifen signal transducer and activator of transcription 3 Mus musculus
6 In the present study, we assessed the biological significance of STAT3 activity in subacute MI using tamoxifen (TM)-inducible cardiac-specific STAT3 knockout (STAT3 iCKO) mice. Tamoxifen signal transducer and activator of transcription 3 Mus musculus