Title : Overproduction of nitric oxide by endothelial cells and macrophages contributes to mitochondrial oxidative stress in adrenocortical cells and adrenal insufficiency during endotoxemia.

Pub. Date : 2015 Jun

PMID : 25744413






1 Functional Relationships(s)
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1 In contrast, administration of the NO donor sodium nitroprusside (SNP) led to mitochondrial oxidative stress and dysfunction in adrenal glands, which resulted in a blunted corticosterone response to ACTH. Corticosterone proopiomelanocortin Homo sapiens