Title : Inhibition of Pro-apoptotic BAX by a noncanonical interaction mechanism.

Pub. Date : 2015 Mar 5

PMID : 25684204






1 Functional Relationships(s)
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1 Hydrogen-deuterium exchange mass spectrometry demonstrated that the N-terminal conformational changes in BAX induced by a triggering BIM BH3 helix were suppressed by the BCL-2 BH4 helix. BH 3 BCL2 associated X, apoptosis regulator Homo sapiens