Pub. Date : 2015 Mar 2
PMID : 25621498
1 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | Preclinical studies suggest that noncytotoxic concentrations of the DNA methyltransferase 1 (DNMT1) inhibitor decitabine produce p53-independent cell-cycle exits by reversing aberrant epigenetic repression of proliferation-terminating (MYC-antagonizing) differentiation genes in cancer cells. | Decitabine | tumor protein p53 | Homo sapiens |