Title : BMI1 attenuates etoposide-induced G2/M checkpoints via reducing ATM activation.

Pub. Date : 2015 Jun 4

PMID : 25088203






1 Functional Relationships(s)
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1 Furthermore, the phosphorylation of ATM targets, including gammaH2AX, threonine 68 (T68) on CHK2 (CHK2 pT68) and serine 15 (S15) on p53 were decreased in overexpression and increased in knockdown BMI1 cells in response to ETOP. Etoposide tumor protein p53 Homo sapiens