Title : Ultrastructural analysis of autophagosome organization using mammalian autophagy-deficient cells.

Pub. Date : 2014 Sep 15

PMID : 25052093






4 Functional Relationships(s)
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1 Here, systematic ultrastructural analysis of mouse embryonic fibroblasts (MEFs) and HeLa cells deficient in various ATG proteins reveals that the emergence of the isolation membrane (phagophore) requires FIP200 (also known as RB1CC1), ATG9A and phosphatidylinositol (PtdIns) 3-kinase activity. Phosphatidylinositols RB1 inducible coiled-coil 1 Homo sapiens
2 Here, systematic ultrastructural analysis of mouse embryonic fibroblasts (MEFs) and HeLa cells deficient in various ATG proteins reveals that the emergence of the isolation membrane (phagophore) requires FIP200 (also known as RB1CC1), ATG9A and phosphatidylinositol (PtdIns) 3-kinase activity. Phosphatidylinositols RB1 inducible coiled-coil 1 Homo sapiens
3 Here, systematic ultrastructural analysis of mouse embryonic fibroblasts (MEFs) and HeLa cells deficient in various ATG proteins reveals that the emergence of the isolation membrane (phagophore) requires FIP200 (also known as RB1CC1), ATG9A and phosphatidylinositol (PtdIns) 3-kinase activity. Phosphatidylinositols RB1 inducible coiled-coil 1 Homo sapiens
4 Here, systematic ultrastructural analysis of mouse embryonic fibroblasts (MEFs) and HeLa cells deficient in various ATG proteins reveals that the emergence of the isolation membrane (phagophore) requires FIP200 (also known as RB1CC1), ATG9A and phosphatidylinositol (PtdIns) 3-kinase activity. Phosphatidylinositols RB1 inducible coiled-coil 1 Homo sapiens