Title : Glucose utilization via glycogen phosphorylase sustains proliferation and prevents premature senescence in cancer cells.

Pub. Date : 2012 Dec 5

PMID : 23177934






1 Functional Relationships(s)
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1 PYGL depletion and the consequent glycogen accumulation led to increased reactive oxygen species (ROS) levels that contributed to a p53-dependent induction of senescence and markedly impaired tumorigenesis in vivo. Glycogen tumor protein p53 Homo sapiens