Title : Oxidized low-density lipoproteins impair endothelial function by inhibiting non-genomic action of thyroid hormone-mediated nitric oxide production in human endothelial cells.

Pub. Date : 2013 Feb

PMID : 23072587






3 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 3,5,3"-triiodothyronine (T(3)), the active form of TH, induces the activation of endothelial nitric oxide synthase via PI3K/AKT non-genomic signaling. Triiodothyronine AKT serine/threonine kinase 1 Homo sapiens
2 3,5,3"-triiodothyronine (T(3)), the active form of TH, induces the activation of endothelial nitric oxide synthase via PI3K/AKT non-genomic signaling. Triiodothyronine AKT serine/threonine kinase 1 Homo sapiens
3 The aim of this study was to investigate the effects of both native LDL (nLDL) and oxLDL on T(3)-mediated AKT phosphorylation, nitric oxide (NO), and cyclic guanosine monophosphate (cGMP) production in human endothelial cells. Triiodothyronine AKT serine/threonine kinase 1 Homo sapiens