Title : Silencing of tomato RBOH1 and MPK2 abolishes brassinosteroid-induced H₂O₂ generation and stress tolerance.

Pub. Date : 2013 Apr

PMID : 22994632






5 Functional Relationships(s)
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1 Here we showed that 24-epibrassinolide (EBR), which induced tolerance to oxidative and heat stress in tomato, was also capable of elevating the transcript levels of RBOH1, MPK1 and MPK2, increasing apoplastic H2 O2 accumulation, and enhancing activation of MPK1/2. brassinolide Mitogen-activated protein kinase Solanum lycopersicum
2 Here we showed that 24-epibrassinolide (EBR), which induced tolerance to oxidative and heat stress in tomato, was also capable of elevating the transcript levels of RBOH1, MPK1 and MPK2, increasing apoplastic H2 O2 accumulation, and enhancing activation of MPK1/2. brassinolide Mitogen-activated protein kinase Solanum lycopersicum
3 Here we showed that 24-epibrassinolide (EBR), which induced tolerance to oxidative and heat stress in tomato, was also capable of elevating the transcript levels of RBOH1, MPK1 and MPK2, increasing apoplastic H2 O2 accumulation, and enhancing activation of MPK1/2. brassinolide Mitogen-activated protein kinase Solanum lycopersicum
4 Here we showed that 24-epibrassinolide (EBR), which induced tolerance to oxidative and heat stress in tomato, was also capable of elevating the transcript levels of RBOH1, MPK1 and MPK2, increasing apoplastic H2 O2 accumulation, and enhancing activation of MPK1/2. brassinolide Mitogen-activated protein kinase Solanum lycopersicum
5 These results suggested that MPK2 played a more critical role than MPK1 in EBR-induced apoplastic H2 O2 accumulation. brassinolide Mitogen-activated protein kinase Solanum lycopersicum