Title : Innate immune activation by inhaled lipopolysaccharide, independent of oxidative stress, exacerbates silica-induced pulmonary fibrosis in mice.

Pub. Date : 2012

PMID : 22815821






1 Functional Relationships(s)
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1 We observed that mice receiving both silica and LPS had significantly more total inflammatory cells, more whole lung lavage MCP-1, MIP-2, KC and IL-1beta, more evidence of oxidative stress and more total lung hydroxyproline than mice receiving either LPS alone, or silica alone. Silicon Dioxide interleukin 1 beta Mus musculus