Title : Glutamate released by Japanese encephalitis virus-infected microglia involves TNF-α signaling and contributes to neuronal death.

Pub. Date : 2012 Mar

PMID : 22144112






2 Functional Relationships(s)
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1 We further demonstrated that tumor necrosis factor-alpha (TNF-alpha) was a key cytokine which stimulated extensive microglial glutamate release by up-regulating glutaminase expression via signals involving protein kinase C, cAMP responsive element-binding protein, and CAAT-enhancer-binding protein-beta. Cyclic AMP tumor necrosis factor Homo sapiens
2 We further demonstrated that tumor necrosis factor-alpha (TNF-alpha) was a key cytokine which stimulated extensive microglial glutamate release by up-regulating glutaminase expression via signals involving protein kinase C, cAMP responsive element-binding protein, and CAAT-enhancer-binding protein-beta. Cyclic AMP tumor necrosis factor Homo sapiens