Title : Influence of experimental subarachnoid hemorrhage on nicotine-induced contraction of the rat basilar artery in relation to nicotinic acetylcholine receptors, calcium, and potassium channels.

Pub. Date : 2013 May

PMID : 22056221






3 Functional Relationships(s)
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1 RESULTS: A central nervous system (CNS) nicotinic acetylcholine receptor (nAChR) and autonomic ganglionic nAChR antagonist (mecamylamine) and skeletal muscle nAChR antagonist (gallamine) concentration-dependently attenuated the nicotine-induced contraction. Nicotine cholinergic receptor nicotinic alpha 2 subunit Rattus norvegicus
2 CONCLUSIONS: Nicotine-induced contraction of the rat basilar artery involved the CNS nAChR subfamily, skeletal muscle nAChR subfamily, and L-type Ca(2+) channel pathways. Nicotine cholinergic receptor nicotinic alpha 2 subunit Rattus norvegicus
3 CONCLUSIONS: Nicotine-induced contraction of the rat basilar artery involved the CNS nAChR subfamily, skeletal muscle nAChR subfamily, and L-type Ca(2+) channel pathways. Nicotine cholinergic receptor nicotinic alpha 2 subunit Rattus norvegicus