Title : Dihydrotestosterone inhibits interleukin-1α or tumor necrosis factor α-induced proinflammatory cytokine production via androgen receptor-dependent inhibition of nuclear factor-κB activation in rheumatoid fibroblast-like synovial cell line.

Pub. Date : 2011

PMID : 22040886






5 Functional Relationships(s)
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1 Dihydrotestosterone inhibits interleukin-1alpha or tumor necrosis factor alpha-induced proinflammatory cytokine production via androgen receptor-dependent inhibition of nuclear factor-kappaB activation in rheumatoid fibroblast-like synovial cell line. Dihydrotestosterone tumor necrosis factor Homo sapiens
2 DHT also inhibited the effects of TNFalpha on the cells overexpressed with AR, indicating that sufficient expression level of functional AR was necessary for the inhibitory effect of DHT on TNFalpha. Dihydrotestosterone tumor necrosis factor Homo sapiens
3 DHT also inhibited the effects of TNFalpha on the cells overexpressed with AR, indicating that sufficient expression level of functional AR was necessary for the inhibitory effect of DHT on TNFalpha. Dihydrotestosterone tumor necrosis factor Homo sapiens
4 DHT also inhibited the effects of TNFalpha on the cells overexpressed with AR, indicating that sufficient expression level of functional AR was necessary for the inhibitory effect of DHT on TNFalpha. Dihydrotestosterone tumor necrosis factor Homo sapiens
5 DHT also inhibited the effects of TNFalpha on the cells overexpressed with AR, indicating that sufficient expression level of functional AR was necessary for the inhibitory effect of DHT on TNFalpha. Dihydrotestosterone tumor necrosis factor Homo sapiens