Title : Prostaglandin E2 and IL-23 plus IL-1β differentially regulate the Th1/Th17 immune response of human CD161(+) CD4(+) memory T cells.

Pub. Date : 2011 Aug

PMID : 21884514






4 Functional Relationships(s)
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1 Prostaglandin E2 (PGE2), interleukin (IL)-23, and IL-1beta (beta) propagate inflammatory bowel disease (IBD) by enhancing the development and function of IL-17 producing CD4(+) T helper (Th17) cells. Dinoprostone interleukin 17A Homo sapiens
2 Prostaglandin E2 (PGE2), interleukin (IL)-23, and IL-1beta (beta) propagate inflammatory bowel disease (IBD) by enhancing the development and function of IL-17 producing CD4(+) T helper (Th17) cells. Dinoprostone interleukin 17A Homo sapiens
3 PGE2 and IL-23 plus IL-1beta synergistically induced early IL-17A secretion from CD161(+) CD4(+) memory T cells and the selective enrichment of IL-17A(+) CD161(+) CD4(+) memory T cells in culture. Dinoprostone interleukin 17A Homo sapiens
4 PGE2 and IL-23 plus IL-1beta synergistically induced early IL-17A secretion from CD161(+) CD4(+) memory T cells and the selective enrichment of IL-17A(+) CD161(+) CD4(+) memory T cells in culture. Dinoprostone interleukin 17A Homo sapiens